Pharmacological Management of Congestive Heart Failure Using Captopril Research Paper

Pharmacological Management of Congestive Heart Failure Using Captopril and Its healing(predicate) Implications (Drug Study ) - Research Paper ExampleThe readings also showed that the pharmacological management of CHF may be typified into beginning(a) line and scrap line agents. The first line agents consist mainly of boom-I, diuretics and beta blockers, while the second line agents consists of digoxin and hydralazine-isosorbide dinitrate. Captropil belongs to the second line of pharmacological agents. Use of Captropil had been found to enhance cardiac performance, among differents. However, ACE inhibitors like Captropil sometimes causes in amigo-edema, painful tongue ulcers, and alveoli is to name a few. The implications of these and the rest of the findings are discussed in the conclusion. Discussion Congestive eye also-ran Congestive tone failure (CHF) is defined as an inability of the heart in supplying normal group O and nutrient demands to the body. CHF squirt be caus ed by a number of affections which (1) weaken the cardiac go across (2) harden the cardiac muscle and (3) increase tissue oxygen demand beyond the capacity of the heart. This condition occurs primarily on elderly patients (Kaufman et al., 2008). Congestive heart failure commonly wills from a dysfunctional heart muscle or myocardial dysfunction. CHF may also result from changes in the structure and function of the heart medically termed as progressive cardiac remodeling (Medifocus, 2011). The heart muscle enables it to pump lineage to facilitate metabolism for the various tissues of the body. When the heart muscles do not function normally, blood flow through the heart and the entire body proceeds slower than normal. This causes increased pressure in the heart. The body responds to the decrease in the hearts pumping capacity by the so-called compensatory mechanisms so that cardiac function can be maintained, such as the renin-angiotensin-aldosterone system and the sympathetic ner vous system (Medifocus, 2011). The compensatory mechanisms can be activated for a period of months or years. During the time that these compensatory systems are functioning, no evident symptoms of heart failure are presented and the patient is regarded as well (Medifocus, 2011). Heart disease classified as asymptomatic is one in which the patient does not experience undue fatigue, dyspnea, palpitations and chest pain during ordinary activities (Capezuti, Siegler, & Mezey, 2008). It should, however, be made very clear at this point that the functioning of the aforementioned compensatory systems will cause boost impairment to the heart in the long run, and consequently affect the circulation of the blood in the body. Such damage is caused by the changes brought about by process of structural remodeling where the heart may enlarge, the cardiac walls may change or become thinner and further decrease in the pumping capacity of the heart. One or a combination of these changes furthe r weakens the heart and causes the pumping to be less effective until the patient eventually develops symptomatic heart failure (Medifocus, 2011). How CHF manifests itself depends on the type of stress the heart is being subjected to, in addition to which ventricle is affected. For example, disease affecting the left ventricle causes pulmonary edema. Meanwhile, disease affecting the right ventricle is more likely to cause edema to the lower limbs and other parts of the body. Moreover, CHF can affect the flow of blood to other organs. For example, inadequate blood flow to